Thioacetals
Sigma Aldrich 2-Bromo-5-(trifluoromethyl)pyridine
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Sigma Aldrich 3-Methyl-3-penten-2-one
MilliporeSigma Sigma Organics products encompass a wide range of quality reagents, solvents, catalysts, and building blocks for organic synthesis. From benchtop discovery to process development and scale-up, Sigma Organics solutions are built to meet the needs of synthetic chemists.
Sigma Aldrich 3-(4-Methoxy-1-naphthyl)acrylic acid
MilliporeSigma Sigma Organics products encompass a wide range of quality reagents, solvents, catalysts, and building blocks for organic synthesis. From benchtop discovery to process development and scale-up, Sigma Organics solutions are built to meet the needs of synthetic chemists.
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Sigma Aldrich 2-Azabicyclo[2.2.1]heptane
MilliporeSigma Sigma Organics products encompass a wide range of quality reagents, solvents, catalysts, and building blocks for organic synthesis. From benchtop discovery to process development and scale-up, Sigma Organics solutions are built to meet the needs of synthetic chemists.
Sigma Aldrich 4-Methylthiophene-2-carboxaldehyde
MilliporeSigma Sigma Organics products encompass a wide range of quality reagents, solvents, catalysts, and building blocks for organic synthesis. From benchtop discovery to process development and scale-up, Sigma Organics solutions are built to meet the needs of synthetic chemists.
Sigma Aldrich Thioacetamide
MilliporeSigma Sigma Organics products encompass a wide range of quality reagents, solvents, catalysts, and building blocks for organic synthesis. From benchtop discovery to process development and scale-up, Sigma Organics solutions are built to meet the needs of synthetic chemists.
Medchemexpress LLC Thioacetamide | 62-55-5 | MFCD00008070 | 98.0% | 75.13 | C2H5NS | 500mg
Thioacetamide (TAA) is an indirect hepatotoxin and causes parenchymal cell necrosis Thioacetamide requires metabolic activation by microsomal CYP2E1 to thioacetamide-S-oxide initially and then to thioacetamide-S-dioxide which is a highly reactive metabolite and its reactive metabolites covalently bind to proteins and lipids thereby causing oxidative stress and centrilobular necrosis Thioacetamide can induce chronic liver fibrosis encephalopathy and other events model[1][2][3][4]
Selleck Chemical LLC Thioacetamide-E0156-25G
Thioacetamide (TAA) is a hepatotoxin frequently used for experimental purposes which produces centrilobular necrosis after a single dose administration Thioacetamide can be used to induce animal models of Cirrhosis
Medchemexpress LLC Thioacetamide | 62-55-5 | MFCD00008070 | 98.0% | 75.13 g/mol | C2H5NS | 1mL
Thioacetamide (TAA) is an indirect hepatotoxin and causes parenchymal cell necrosis Thioacetamide requires metabolic activation by microsomal CYP2E1 to thioacetamide-S-oxide initially and then to thioacetamide-S-dioxide which is a highly reactive metabolite and its reactive metabolites covalently bind to proteins and lipids thereby causing oxidative stress and centrilobular necrosis Thioacetamide can induce chronic liver fibrosis encephalopathy and other events model[1][2][3][4]
ALADDIN SCIENTIFIC CORPORATION Thioacetamide, 25g
Thioacetamide is a thiocarbonyl compound It undergoes deuterium exchange reaction with D2O to afford N-dideuterated thioacetamide Infrared spectral investigations of thioacetamide and its deuterated derivative (N-dideuterated thioacetamide) have been reported Usually used as Hepatotoxin and carcinogenand also used as an additive in enantioselective reduction of -keto esters with immobilized baker s yeast Thioacetamide (TAA) is an indirect hepatotoxin and causes parenchymal cell necrosis Thioacetamide requires metabolic activation by microsomal CYP2E1 to thioacetamide-S-oxide initially and then to thioacetamide-S-dioxide which is a highly reactive metabolite and its reactive metabolites covalently bind to proteins and lipids thereby causing oxidative stress and centrilobular necrosis Thioacetamide can induce chronic liver fibrosis encephalopathy and other events model
Medchemexpress LLC Thioacetamide | 62-55-5 | MFCD00008070 | 98.0% | 75.13 g/mol | C2H5NS | 1g
Thioacetamide (TAA) is an indirect hepatotoxin and causes parenchymal cell necrosis Thioacetamide requires metabolic activation by microsomal CYP2E1 to thioacetamide-S-oxide initially and then to thioacetamide-S-dioxide which is a highly reactive metabolite and its reactive metabolites covalently bind to proteins and lipids thereby causing oxidative stress and centrilobular necrosis Thioacetamide can induce chronic liver fibrosis encephalopathy and other events model[1][2][3][4]