Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

Cadherin-6/KCAD Antibody (2B6), Alexa Fluor™ 532, Novus Biologicals™
SDP

Mouse Monoclonal Antibody

Supplier:  Novus Biologicals NBP197689AF532

Catalog No. NP197689A32


Only null left
Add to Cart

Description

Description

Cadherin-6/KCAD Monoclonal specifically detects Cadherin-6/KCAD in Human, Rat samples. It is validated for Western Blot, Immunohistochemistry, Immunocytochemistry/Immunofluorescence, Immunohistochemistry-Paraffin, Immunohistochemistry-Frozen.
Specifications

Specifications

Cadherin-6/KCAD
Monoclonal
Alexa Fluor 532
50 mM sodium borate with 0.05% sodium azide
CDH6
2B6 is a mouse monoclonal IgG1 antibody obtained by fusion of SP2/0 mouse myeloma cells with spleen cells from a BALB/c mouse immunized with affinity purified extracellular domain of Human cadherin-6-GST fusion protein.
Primary
2B6 recognizes the extracellular domain of cadherin-6.
Store at 4°C in the dark.
Western Blot, Immunohistochemistry, Immunocytochemistry, Immunofluorescence, Immunohistochemistry (Paraffin)
2B6
Western Blot, Immunohistochemistry, Immunocytochemistry/Immunofluorescence, Immunohistochemistry-Paraffin, Immunohistochemistry-Frozen
cadherin 6, type 2, K-cadherin (fetal kidney), cadherin, fetal kidney, cadherin-6, KCAD, K-cadherin, K-cadherin (fetal kidney)
Mouse
0.1 mL
1004
Human, Rat
IgG1
Product Suggestions

Product Suggestions

Videos
SDS
Documents

Documents

Product Certifications
Promotions

Promotions

Product Content Correction

Your input is important to us. Please complete this form to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Your feedback has been submitted: Thank you for helping us improve our website.