Carbohydrates
Medchemexpress LLC D(+)-Galactosamine hydrochloride | 1772-03-8 | 99.9% | 215.63 | 100 MG
D(+)-Galactosamine hydrochloride | 1772-03-8 | 99.9% | 215.63 | 100 MG
Medchemexpress LLC D(+)-Galactosamine hydrochloride | 1772-03-8 | 99.9% | 215.63 | 25 MG
D(+)-Galactosamine hydrochloride is an established experimental toxin that primarily causes liver injury through the generation of free radicals and depletion of UTP nucleotides. Intoxication with D(+)-Galactosamine hydrochloride also induces renal dysfunction, with renal failure often associated with the end-stage of liver damage. The combination of Lipopolysaccharide/D(+)-Galactosamine is a known animal model for inducing acute liver injury, leading to fulminant hepatic failure.
- Causes liver injury through generation of free radicals.
- Leads to depletion of UTP nucleotides.
- Induces renal dysfunction.
- Useful in animal models for acute liver injury.
Medchemexpress LLC O-Linked GlcNAc transferase substrate | 954381-93-2 | 98.25% | 1783.08 | 10 MG
O-Linked GlcNAc transferase substrate is a biologically active peptide that functions as a substrate for O-linked GlcNAc transferase (OGT), a eukaryotic glycosyltransferase that utilizes UDP-GlcNAc as a glycosyl donor.
- Biological active peptide
- Substrate of O-linked GlcNAc transferase (OGT)
- White to off-white solid appearance
- High purity of 98.25%
- Soluble in DMSO and H2O at 100 mg/mL
- Sequence: Lys-Lys-Lys-Tyr-Pro-Gly-Gly-Ser-Thr-Pro-Val-Ser-Ser-Ala-Asn-Met-Met
Sigma Aldrich Fine Chemicals Biosciences Hyaluronic acid sodium salt from Streptococcus equi mol wt 2,000,000-2,400,000 | 9067-32-7 | MFCD01773053 | 50MG
Hyaluronic acid sodium salt from Streptococcus equi mol wt 2,000,000-2,400,000 | 9067-32-7 | MFCD01773053 | 50MG
Medchemexpress LLC D(+)-Galactosamine hydrochloride | 1772-03-8 | 99.9% | 215.63 | 1 G
D(+)-Galactosamine hydrochloride is an established experimental toxin known to primarily cause liver injury by generating free radicals and depleting UTP nucleotides. It also induces renal dysfunction, often leading to renal failure associated with end-stage liver damage. This compound can be used to induce hepatitis models, mimicking viral hepatitis by causing lipid peroxidation and cell membrane deterioration.
- Induces apoptosis and necrosis in primary cultures of rat hepatocytes.
- Causes caspase-3 activation and DNA fragmentation.
- Used as an animal model for fulminant hepatic failure.
- Induces acute liver failure in mice when co-administered with Lipopolysaccharide.
Sigma Aldrich Fine Chemicals Biosciences Hyaluronic acid sodium salt from Streptococcus equi mol wt 10,000-30,000 | 9067-32-7 | MFCD01773053 | 50MG
Hyaluronic acid sodium salt from Streptococcus equi mol wt 10,000-30,000 | 9067-32-7 | MFCD01773053 | 50MG
Sigma Aldrich Fine Chemicals Biosciences Hyaluronic acid sodium salt from bovine vitreous humor | 9067-32-7 | MFCD01773053 | 10MG
Hyaluronic acid sodium salt from bovine vitreous humor | 9067-32-7 | MFCD01773053 | 10MG
Apexbio Technology LLC N-acetyl D-galactosamine 1811-31-0 100mg
N-acetyl D-galactosamine (CAS 1811-31-0) is an endogenous amino sugar that serves as a structural component in glycoproteins and glycolipids It functions as a fundamental building block in the biosynthesis of O-linked glycoproteins participating in post-translational modification and influencing cell signaling adhesion and molecular recognition processes Due to its role in the formation of brain heteropolysaccharides N-acetyl D-galactosamine is frequently utilized in studies investigating glycosylation neural development and carbohydrate-mediated cellular interactions within biomedical research
Sigma Aldrich Fine Chemicals Biosciences alpha D Glucosamine 1 phos25MG
alpha D Glucosamine 1 phos25MG
Medchemexpress LLC D(+)-Galactosamine hydrochloride | 1772-03-8 | 99.9% | 215.63 | 500 MG
D(+)-Galactosamine hydrochloride is an established experimental toxin that primarily causes liver injury by generating free radicals and depleting UTP nucleotides. It also induces renal dysfunction, with renal failure often associated with the end-stage of liver damage. Lipopolysaccharide/D(+)-Galactosamine-induced acute liver injury is a known animal model of fulminant hepatic failure.
- Induces apoptosis and necrosis in primary rat hepatocyte cultures.
- Causes caspase-3 activation and DNA fragmentation.
- Induces hepatotoxicity similar to viral hepatitis.
- Suitable for research use only.
Bioworld N-ACETYL-GALACTOSAMINE SEPA
N-ACETYL-GALACTOSAMINE SEPAROPORE (AGAROSE): N-acetyl-galactosamine Separopore n-acetyl-galactosamine is conjugated to Separopore.
Sigma Aldrich Fine Chemicals Biosciences L-(+)-Arabinose | 5328-37-0 | MFCD00135866 | Sample
L-(+)-Arabinose | Purity: 99% | Mol Wt: 150.13 | 5328-37-0 | MFCD00135866 | Sample